Disabled Vet for decades suffers from all of the above and can only sleep on one side barely, 20 minute intervals taking up to hours to fall back asleep for another 20 minute interval. Not looking for pity just tips and hints and opinions. Tried most everything been at this most of my life,,, Willing to listen! Tollorant of all and I mean all opiates. Thats what brought down my testosterone levels . Been on hormone therapy for only a week, have not seen change yet? take topically once a day. Doc says for taking pain meds for so long that my testosterone is way down. and antidepressants won’t have an effect if any at all. Which by the way have tried about 15-17 different kinds over the years. I am immune to any and all narcotics now a days no matter the strength and I have done everything,,, I can’t sleep ever , have no social life and don’t care much about it at times ,,,no friends,,,no interest in anything anymore,,,only leave to go to the doctor and not even that sometimes.




do you regret doing it? are there better ways to increase your testosterone levels? natural ways? Negative side effects? Are you concerned about the dangers? Does it really make a big difference in your mood, sex drive, penis function, etc? Thanks.

hormonal imbalance questions..?




so my doctor said my testosterone levels are high so i have a hormonal imbalance. my period is late by like a week and i have been cramping BAD BAD BAD BAAAAD! I never cramp but when i have its never been like this. My stomach is killing me to. Like im about to puke. Explain this stuff with ‘hormones’




Do you grow in height? Do your testosterone levels get higher?

Anything else?

guys who have had hormone therapy, please answer?




What were the effects you noticed when they finally kicked in? I’m a young man who needs hormone therapy due to an underactive pituatary. My testosterone levels were checked and they were very low. I also think my growth hormones are low because i never heal from tissue injuries.

I am having alot of joint pain and my sex drive is that of a 70 year old man. If you have had hormone therapy and similar problems, how are you now with the therapy?




http://www.youtube.com/watch?v=cm5inkRIKI4&feature=channel_page

What do you think about this? Wait til 2:00, that’s the good part, where he touches on feminism.

During menopause, women’s estrogen levels drop and their testosterone levels rise — that’s why women in their 40′s begin to have an increase of facial hair and body hair, and hair starts to thin in their scalps.

So, when I turn 40, and my estrogen levels rise, will my voice get higher pitched? Will I start eating chocolate ice cream out of the pint, while watching Oprah and talking on the phone twirling on the cord and kicking my leg up in the air? Will I start to complain & whine more? Will I go on G&WS and bash men?

What exactly are the effects of male menopause?

The second half of the video, the doctor gives advice on how we should prevent this. Simply put, like most things in life — nutrition and exercise. We can prevent the rise in estrogen and keep our testosterone levels high if we follow his steps. I will definitely heed to his advice. Heaven forbid I become estrogen-laden and end up like some of the women on this forum.







The following is provided for information purposes only. If you believe you have a medical condition requiring treatment, please consult a qualified physician.

Two years ago, my doctor ordered routine blood tests as a part of my yearly check-up, and the lab mistakenly measured 17-alpha hydroxyprogesterone (17-OHP). Although I am an adult male, my 17-OHP level is in the prepubertal range without evidence of a clinical disease.

17-OHP is considered to be a precursor of both testosterone (T) and cortisol. It is rarely, if ever, measured in men, and is primarily evaluated during pregnancy, or during the diagnostic workup for female infertility and congenital adrenal hyperplasia in children. If a man’s T level is normal, 17-OHP is assumed to be normal. Although my testosterone (T) level is normal, 17-OHP is abnormal and my ratio of 17-OHP to T is 0.02. For men with normal testicular function the ratio is 0.24 +/- 0.08 (J Clin Endocrinol Metab. 1978 Nov;47(5):1144-7; Basal and human chorionic gonadotropin-stimulated 17 alpha-hydroxyprogesterone and testosterone levels in Klinefelter’s syndrome).

Low 17-OHP levels are associated with combat stress, old age, steroid abuse; disorders such as Addison’s disease, adrenal hypoplasia congenita, adrenal exhaustion, hypogonadism; and various intersex disorders such 17-beta hydroxysteroid dehydrogenase deficiency and Klinefelter’s syndrome, a genetic condition characterized by an XXY chromosome pattern.

Both T and 17-OHP dramatically increase in males before and after birth. During the first 1-2 months of life, these hormones surge to adult levels during a period know as the “mini puberty” of infancy. Research suggests that exposure to prenatal stress can disrupted the surge in these hormones during critical phases of brain development.

Studies show that exogenous T increases a man’s sex drive without changing his sexual orientation. Although it is well established that T is necessary for the development of male secondary sex characteristics, low, high or normal T levels alone do not determine a man’s sexual orientation.

Again, it is interesting to note that both T AND 17-OHP surge simultaneously during the prenatal, postnatal, and adolescent periods of male sexual development. This seems to suggest that T may work together with either 17-OHP or some other hormone for which 17-OHP is a precursor, perhaps epitestosterone (EpiT). Too much or too little T in the absence of corresponding levels 17-OHP or EpiT in the blood may result in a homosexual orientation. It is possible that an imbalance between 17-OHP and T or T and EpiT may distinguish homosexual from heterosexual men. Currently, EpiT is only measured in urine for the purpose of detect illicit anabolic steroid use by athletes.

I worked with my doctor for a year before finding that the vitamin that balances my hormones is pyridoxine or vitamin B6, often referred to as the “anti-stress vitamin.” Although my vitamin B6 level is normal, B6 at 150-200 mg per day for one month normalizes my 17-OHP level, while raising the ratio of 17-OHP to T from a baseline of 0.02 to 0.17. However, after a month without therapy my levels return to baseline.

It is well established that steroid hormones, such as estrogen and testosterone, exert their effects in the body by binding to steroid hormone receptors in the nucleus of the cell and altering gene transcription. Interestingly, the bioactive form of vitamin B6, pyridoxal-5-phosphate (PLP) binds to steroid receptors in a manner that inhibits the binding of steroid hormones, thus decreasing their effects. Consequently, increased binding of PLP to steroid receptors for estrogen, progesterone, testosterone, and other steroid hormones may explain why mega doses vitamin B6 correct my hormone imbalance.

I encourage anyone who believes there is a biological basis for his same-sex attraction to have his T, progesterone and 17-OHP levels measured. If you are unable to find a doctor to do so, or privacy is an issue, order the tests yourself through a direct access laboratory such as EconoLabs or Health Tests Direct.

My doctor always obtained both baseline and post-therapy early morning (8:00 AM), fasting blood samples. Recent vitamin, mineral, and prescription drug intake may compromise the accuracy of results. I eliminated them from my diet for at least two weeks prior to any blood test.

The Postnatal Gonadotropin and Sex Steroid Surge—Insights from the Androgen Insensitivity Syndrome The Journal of Clinical Endocrinology & Metabolism Vol. 87, No. 1 24-28

http://jcem.endojournals.org/cgi/content/short/87/1/24

Homosexuality is not a disease. Many heterosexuals also experience same-sex attraction. There are three questions here:

1) Does vitamin B6 influence hormone levels?

2) Do 17-OHP levels and/or 17-OHP to T ratios distinguish homosexual from heterosexual males?

3) If such a difference exists, does vitamin B6 influence hormone levels and/or brain regions associated with sexual orientation?







The following is provided for information purposes only. If you believe you have a medical condition requiring treatment, please consult a qualified physician.

Two years ago, my doctor ordered routine blood tests as a part of my yearly check-up, and the lab mistakenly measured 17-alpha hydroxyprogesterone (17-OHP). Although I am an adult male, my 17-OHP level is in the prepubertal range without evidence of a clinical disease.

17-OHP is considered to be a precursor of both testosterone (T) and cortisol. It is rarely, if ever, measured in men, and is primarily evaluated during pregnancy, or during the diagnostic workup for female infertility and congenital adrenal hyperplasia in children. If a man’s T level is normal, 17-OHP is assumed to be normal. Although my testosterone (T) level is normal, 17-OHP is abnormal and my ratio of 17-OHP to T is 0.02. For men with normal testicular function the ratio is 0.24 +/- 0.08 (J Clin Endocrinol Metab. 1978 Nov;47(5):1144-7; Basal and human chorionic gonadotropin-stimulated 17 alpha-hydroxyprogesterone and testosterone levels in Klinefelter’s syndrome).

Low 17-OHP levels are associated with combat stress, old age, steroid abuse; disorders such as Addison’s disease, adrenal hypoplasia congenita, adrenal exhaustion, hypogonadism; and various intersex disorders such 17-beta hydroxysteroid dehydrogenase deficiency and Klinefelter’s syndrome, a genetic condition characterized by an XXY chromosome pattern.

Both T and 17-OHP dramatically increase in males before and after birth. During the first 1-2 months of life, these hormones surge to adult levels during a period know as the “mini puberty” of infancy. Research suggests that exposure to prenatal stress can disrupted the surge in these hormones during critical phases of brain development.

Studies show that exogenous T increases a man’s sex drive without changing his sexual orientation. Although it is well established that T is necessary for the development of male secondary sex characteristics, low, high or normal T levels alone do not determine a man’s sexual orientation.

Again, it is interesting to note that both T AND 17-OHP surge simultaneously during the prenatal, postnatal, and adolescent periods of male sexual development. This seems to suggest that T may work together with either 17-OHP or some other hormone for which 17-OHP is a precursor, perhaps epitestosterone (EpiT). Too much or too little T in the absence of corresponding levels 17-OHP or EpiT in the blood may result in a homosexual orientation. It is possible that an imbalance between 17-OHP and T or T and EpiT may distinguish homosexual from heterosexual men. Currently, EpiT is only measured in urine for the purpose of detect illicit anabolic steroid use by athletes.

I worked with my doctor for a year before finding that the vitamin that balances my hormones is pyridoxine or vitamin B6, often referred to as the “anti-stress vitamin.” Although my vitamin B6 level is normal, B6 at 150-200 mg per day for one month normalizes my 17-OHP level, while raising the ratio of 17-OHP to T from a baseline of 0.02 to 0.17. However, after a month without therapy my levels return to baseline.

It is well established that steroid hormones, such as estrogen and testosterone, exert their effects in the body by binding to steroid hormone receptors in the nucleus of the cell and altering gene transcription. Interestingly, the bioactive form of vitamin B6, pyridoxal-5-phosphate (PLP) binds to steroid receptors in a manner that inhibits the binding of steroid hormones, thus decreasing their effects. Consequently, increased binding of PLP to steroid receptors for estrogen, progesterone, testosterone, and other steroid hormones may explain why mega doses vitamin B6 correct my hormone imbalance.

I encourage anyone who believes there is a biological basis for his same-sex attraction to have his T, progesterone and 17-OHP levels measured. If you are unable to find a doctor to do so, or privacy is an issue, order the tests yourself through a direct access laboratory such as EconoLabs or Health Tests Direct.

My doctor always obtained both baseline and post-therapy early morning (8:00 AM), fasting blood samples. Recent vitamin, mineral, and prescription drug intake may compromise the accuracy of results. I eliminated them from my diet for at least two weeks prior to any blood test.

The Postnatal Gonadotropin and Sex Steroid Surge—Insights from the Androgen Insensitivity Syndrome The Journal of Clinical Endocrinology & Metabolism Vol. 87, No. 1 24-28

http://jcem.endojournals.org/cgi/content/short/87/1/24

To date, there are no published, peer-reviewed studies of 17-OHP levels or 17-OHP to T ratios in homosexual males.
Homosexuality is not a disease. Many heterosexuals also experience same-sex attraction. There are three questions here:

1) Does vitamin B6 influence hormone levels?

2) Do 17-OHP levels and/or 17-OHP to T ratios distinguish homosexual from heterosexual males?

3) If such a difference exists, does vitamin B6 influence hormone levels and/or brain regions associated with sexual orientation?

There are no published studies on the effect of vitamins on sexual orientation.




The following is provided for information purposes only. If you believe you have a medical condition requiring treatment, please consult a qualified physician.

Two years ago, my doctor ordered routine blood tests as a part of my yearly check-up, and the lab mistakenly measured 17-alpha hydroxyprogesterone (17-OHP). Although I am an adult male, my 17-OHP level is in the prepubertal range without evidence of a clinical disease.

17-OHP is considered to be a precursor of both testosterone (T) and cortisol. It is rarely, if ever, measured in men, and is primarily evaluated during pregnancy, or during the diagnostic workup for female infertility and congenital adrenal hyperplasia in children. If a man’s T level is normal, 17-OHP is assumed to be normal. Although my testosterone (T) level is normal, 17-OHP is abnormal and my ratio of 17-OHP to T is 0.02. For men with normal testicular function the ratio is 0.24 +/- 0.08 (J Clin Endocrinol Metab. 1978 Nov;47(5):1144-7; Basal and human chorionic gonadotropin-stimulated 17 alpha-hydroxyprogesterone and testosterone levels in Klinefelter’s syndrome).

Low 17-OHP levels are associated with combat stress, old age, steroid abuse; disorders such as Addison’s disease, adrenal hypoplasia congenita, adrenal exhaustion, hypogonadism; and various intersex disorders such 17-beta hydroxysteroid dehydrogenase deficiency and Klinefelter’s syndrome, a genetic condition characterized by an XXY chromosome pattern.

Both T and 17-OHP dramatically increase in males before and after birth. During the first 1-2 months of life, these hormones surge to adult levels during a period know as the “mini puberty” of infancy. Research suggests that exposure to prenatal stress can disrupted the surge in these hormones during critical phases of brain development.

Studies show that exogenous T increases a man’s sex drive without changing his sexual orientation. Although it is well established that T is necessary for the development of male secondary sex characteristics, low, high or normal T levels alone do not determine a man’s sexual orientation.

Again, it is interesting to note that both T AND 17-OHP surge simultaneously during the prenatal, postnatal, and adolescent periods of male sexual development. This seems to suggest that T may work together with either 17-OHP or some other hormone for which 17-OHP is a precursor, perhaps epitestosterone (EpiT). Too much or too little T in the absence of corresponding levels 17-OHP or EpiT in the blood may result in a homosexual orientation. It is possible that an imbalance between 17-OHP and T or T and EpiT may distinguish homosexual from heterosexual men. Currently, EpiT is only measured in urine for the purpose of detect illicit anabolic steroid use by athletes.

I worked with my doctor for a year before finding that the vitamin that balances my hormones is pyridoxine or vitamin B6, often referred to as the “anti-stress vitamin.” Although my vitamin B6 level is normal, B6 at 150-200 mg per day for one month normalizes my 17-OHP level, while raising the ratio of 17-OHP to T from a baseline of 0.02 to 0.17. However, after a month without therapy my levels return to baseline.

It is well established that steroid hormones, such as estrogen and testosterone, exert their effects in the body by binding to steroid hormone receptors in the nucleus of the cell and altering gene transcription. Interestingly, the bioactive form of vitamin B6, pyridoxal-5-phosphate (PLP) binds to steroid receptors in a manner that inhibits the binding of steroid hormones, thus decreasing their effects. Consequently, increased binding of PLP to steroid receptors for estrogen, progesterone, testosterone, and other steroid hormones may explain why mega doses vitamin B6 correct my hormone imbalance.

I encourage anyone who believes there is a biological basis for his same-sex attraction to have his T, progesterone and 17-OHP levels measured. If you are unable to find a doctor to do so, or privacy is an issue, order the tests yourself through a direct access laboratory such as EconoLabs or Health Tests Direct.

My doctor always obtained both baseline and post-therapy early morning (8:00 AM), fasting blood samples. Recent vitamin, mineral, and prescription drug intake may compromise the accuracy of results. I eliminated them from my diet for at least two weeks prior to any blood test.

The Postnatal Gonadotropin and Sex Steroid Surge—Insights from the Androgen Insensitivity Syndrome The Journal of Clinical Endocrinology & Metabolism Vol. 87, No. 1 24-28

http://jcem.endojournals.org/cgi/content/short/87/1/24

To date, there are no published, peer-reviewed studies of 17-OHP levels or 17-OHP to T ratios in homosexual males.
Homosexuality is not a disease. Many heterosexuals also experience same-sex attraction. There are three questions here:

1) Does vitamin B6 influence hormone levels?

2) Do 17-OHP levels and/or 17-OHP to T ratios distinguish homosexual from heterosexual males?

3) If such a difference exists, does vitamin B6 influence hormone levels and/or brain regions associated with sexual orientation?

There are no published studies on the effect of vitamins on sexual orientation.




The following is provided for information purposes only. If you believe you have a medical condition requiring treatment, please consult a qualified physician.

Two years ago, my doctor ordered routine blood tests as a part of my yearly check-up, and the lab mistakenly measured 17-alpha hydroxyprogesterone (17-OHP). Although I am an adult male, my 17-OHP level is in the prepubertal range without evidence of a clinical disease.

17-OHP is considered to be a precursor of both testosterone (T) and cortisol. It is rarely, if ever, measured in men, and is primarily evaluated during pregnancy, or during the diagnostic workup for female infertility and congenital adrenal hyperplasia in children. If a man’s T level is normal, 17-OHP is assumed to be normal. Although my testosterone (T) level is normal, 17-OHP is abnormal and my ratio of 17-OHP to T is 0.02. For men with normal testicular function the ratio is 0.24 +/- 0.08 (J Clin Endocrinol Metab. 1978 Nov;47(5):1144-7; Basal and human chorionic gonadotropin-stimulated 17 alpha-hydroxyprogesterone and testosterone levels in Klinefelter’s syndrome).

Low 17-OHP levels are associated with combat stress, old age, steroid abuse; disorders such as Addison’s disease, adrenal hypoplasia congenita, adrenal exhaustion, hypogonadism; and various intersex disorders such 17-beta hydroxysteroid dehydrogenase deficiency and Klinefelter’s syndrome, a genetic condition characterized by XXY chromosome pattern.

Both T and 17-OHP dramatically increase in males before and after birth. During the first 1-2 months of life, these hormones surge to adult levels during a period know as the “mini puberty” of infancy. Research suggests that exposure to prenatal stress can disrupted the surge in these hormones during critical phases of brain development.

Studies show that exogenous T increases a man’s sex drive without changing his sexual orientation. Although it is well established that T is necessary for the development of male secondary sex characteristics, low, high or normal T levels alone do not determine a man’s sexual orientation.

Again, it is interesting to note that both T AND 17-OHP surge simultaneously during the prenatal, postnatal, and adolescent periods of male sexual development. This seems to suggest that T may work together with either 17-OHP or some other hormone for which 17-OHP is a precursor, perhaps epitestosterone (EpiT). Too much or too little T in the absence of corresponding levels 17-OHP or EpiT in the blood may result in a homosexual orientation. It is possible that an imbalance between 17-OHP and T or T and EpiT may distinguish homosexual from heterosexual men. Currently, EpiT is only measured in urine for the purpose of detect illicit anabolic steroid use by athletes.

I worked with my doctor for a year before finding that the vitamin that balances my hormones is pyridoxine or vitamin B6, often referred to as the “anti-stress vitamin.” Although my vitamin B6 level is normal, B6 at 150-200 mg per day for one month normalizes my 17-OHP level, while raising the ratio of 17-OHP to T from a baseline of 0.02 to 0.17. However, after a month without therapy my levels return to baseline.

It is well established that steroid hormones, such as estrogen and testosterone, exert their effects in the body by binding to steroid hormone receptors in the nucleus of the cell and altering gene transcription. Interestingly, the bioactive form of vitamin B6, pyridoxal-5-phosphate (PLP) binds to steroid receptors in a manner that inhibits the binding of steroid hormones, thus decreasing their effects. Consequently, increased binding of PLP to steroid receptors for estrogen, progesterone, testosterone, and other steroid hormones may explain why mega doses vitamin B6 correct my hormone imbalance.

I encourage anyone who believes there is a biological basis for his same-sex attraction to have his T, progesterone and 17-OHP levels measured. If you are unable to find a doctor to do so, or privacy is an issue, order the tests yourself through a direct access laboratory such as EconoLabs or Health Tests Direct.

My doctor always obtained both baseline and post-therapy early morning (8:00 AM), fasting blood samples. Recent vitamin, mineral, and prescription drug intake may compromise the accuracy of results. I eliminated them from my diet for at least two weeks prior to any blood test.

The Postnatal Gonadotropin and Sex Steroid Surge—Insights from the Androgen Insensitivity Syndrome The Journal of Clinical Endocrinology & Metabolism Vol. 87, No. 1 24-28

http://jcem.endojournals.org/cgi/content/short/87/1/24

To date, there are no published, peer-reviewed studies of 17-OHP levels and 17-OHP to T ratios in homosexual males.
Homosexuality is not a disease. Many heterosexuals also experience same-sex attraction. There are three questions here:

1) Does vitamin B6 influence hormone levels?

2) Do 17-OHP levels and/or 17-OHP to T ratios distinguish homosexual from heterosexual males?

3) If such a difference exists, does vitamin B6 influence hormone levels and/or brain regions associated with sexual orientation?
There are no published studies on the effect of vitamins on sexual orientation.
Androsterone/Etiocholanolone Ratios in Male Homosexuals

http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1586258&blobtype=pdf